Birth Control

 

 

Did you know that there is a guaranteed 100% effective birth control method and that man has known it since Adam and Eve?  This well-known method works at preventing pregnancy with absolute certainty every time that it is tried.  Are you ready?  It is called abstinence!!!  Yes indeed, abstinence works at preventing pregnancy every time that it is practiced. 

 

Now, if you are happily married, then you do not wish to perpetually abstain.  There are basically two types of birth control methods, natural and chemical.  The best natural method is called the symptothermal method.  Consult the World Health Organization for their publications on this and other natural methods.  If you choose chemical birth control methods then you are more than likely killing your offspring through chemically induced micro abortions. 

 

No, Planned Parenthood along with your liberal teachers and media talking heads did not tell you that chemical contraceptives cause abortions.  One does not live long in life before one learns that there is a polemic distinction between the widely dispensed propaganda of the general public and the real facts of the research community.  We in America have no excuse for not doing our homework.

 

Please take note of the date of the following information.  Can you trust your information sources to tell you the truth on this topic?  If not, then promptly abandon them.

 

The following excerpts are from a book compiled during a Comprehensive Endocrinology Series edited by Giuseppe Benagiano, M.D. and Egon Diczfalusy, M.D., entitled, “Endocrine Mechanisms in Fertility Regulation,” (1983) by Raven Press Books. 

 

Page 7-8:  “Implantation:  Recent evidence indicates that the following events associated with implantation are prostaglandin-related: (a) fluid accumulation by the blastocysts; (b) change in permeability of maternal capillaries near the blastocysts; (c) decidual reaction and accumulation of prostacyclin in the myometrium at the site of implantation.  Since prostaglandins appear to play a role in these and other components of the implantation process, prostaglandin antagonists or inhibitors could be useful contraceptives.  Medicated IUDs releasing prostaglandin inhibitors may be a useful form of contraception.  Further, antiprostaglandins suppress uterine motility and decrease menstrual cramps, suggesting that an improvement in IUD retention rates with a decrease in IUD-related pelvic pain may result from antiprostaglandin-medicated IUDs.

 

A number of substances which either inhibit implantation or interrupt pregnancy after implantation have been investigated over the past decade, and a number of terms have been used to describe these compounds.  These include anti-implantation agents, postcoital contraceptives, morning-after pills, once-a-week pills, interceptive agents, abortifacients, etc.  Estrogens such as diethylstilbestrol, ethinyl estradiol, and conjugated estrogens prevent implantation when high does are used for up to 5 days in women starting within 72 hr of coitus.  However, intensive programs have centered around the search for nonestrogenic preimplantative contragestational agents because of estrogen-related side effects (14,25,27,34,37).

 

Since Corbin and Beattie’s initial report on the abortive effects of administering large doses of GnRH to pregnant female rats (22), a number of confirmatory observations have appeared, showing that 100% inhibition of pregnancy termination occurs in rats treated with as little as 1 mg GnRH per day from days 1 to 7 of pregnancy.  In pregnant rabbits, higher doses of GnRH appear to cause luteolysis and decreased fetal survival.  Since studies of successful pregnancy disruption or prevention of implantation have been associated with decreases in both serum progesterone and estradiol concentrations, it is tempting to speculate that GnRH-medicated luteolysis may cause pregnancy termination.  Evidence for this hypothesis is derived from studies where the antinidatory effect of large doses of GnRH appears to be partially prevented by administration of both progesterone and estrogens.”

 

Page 10:  “Conclusion:  The family of GnRH agonist and antagonist peptides are now in widespread clinical trials, evaluating a variety of reproductive processes: (a) follicular maturation; (b) luteolysis; (c) pituitary gonadotropin secretion; (d) ovulation; (e) implantation; and (f) abortion.

 

…if GnRH can overcome the luteotropic effect of hCG and consequently prevent implantation or interrupt pregnancy…

 

…© disruption of the implanted blastocysts.”

 

Page 19:  “Postcoital Contraception:  It is well documented that the administration of sex hormones immediately postcoitum in an otherwise unprotected intercourse has a good contraceptive effect.  More recently, there have been studies of estrogen-progestogen combinations, administered in the form of commercial Oral Contraceptives…rendering the uterine endometrium unsuitable for implantation.”

 

Page 36:  “Secondary Contraceptive Mechanisms:  Uterine Endometrium:  Although suppression of ovulation is the principal mode of action of all combined Oral Contraceptives, secondary antifertility actions occur at other sites, reducing the chances of conception should an escape ovulation occur.  One of the most important of these secondary effects is the development of a uterine endometrium unsuitable for implantation (103).

 

The cyclic pattern of development of the human endometrium is well known. 

 

Many fixed dose combined Oral Contraceptives completely disrupt the usual cyclic changes in the endometrium.”

 

Page 38:  “Ovum Transport:  A detailed review on ovum transport through the fallopian tubes to the uterus and its role in fertility and contraception has been published (68).  Fertilization of an ovum by a spermatozoan occurs in the oviduct, whereas transport of spermatozoa, unfertilized ova, and fertilized ova through the oviduct depend on tubal motility and integrity.  It is possible that Oral Contraceptive effects on the fallopian tube may contribute to secondary contraceptive effects, though for postcoital hormones this may be a principal effect (177)

 

Significant alterations in the human oviducts following use of combined Oral Contraceptives have been reported by several research groups (8,22,46,53).  Changes in zygote transport could be influenced by alterations in tubal musculature, oviductal fluids, cilial activity, or a combination of these.  The presence of sex hormone receptors in the human oviduct (130) may influence any of these physiological aspects.”

 

Page 40:  “Conclusions: Combined Oral Contraceptives have secondary contraceptive effects, mediated via sex hormone receptors, on the uterine endometrium, cervical mucus, and oviducts; they may also interfere with spermatozoan transport and capacitation in the female tract.”

 

The following is from the “Symposium on Development Of Preimplantation Embryos And Their Environment” (1988: Kyoto, Japan) edited by Koji Yoshinaga and Takahide Mori, Alan R. Liss, Inc. Publisher.

 

Page 289:  “Antiprogestins And Egg-Implantation:  The experimental work reported here deals with PREVENTION of egg-implantation (Intervention) in rats by two such compounds, RU 486 (mifepristone) developed by Roussel-Uclaf and ZK 98734 developed by Schering…”

 

“We have recently shown that mifepristone, administered post-coitally in rats, exerts an antiimplantation effect proportional to the dose administered.”

 

Page 290:  “In the latter studies, mifepristone, administered to rats on days 1 and 2 or day 2 post coitum, was found to induce an obvious acceleration of tubal egg transport as well as a detrimental effect on the prenidatory development of the fertilized egg and its maintenance in utero.”

 

Page 292:  “Mifepristone as well as ZK 98734 induce a delay in endometrial development, as shown by the “out of phase” aspect of the luminal surface of the uterine epithelium, when studied under scanning electron microscopy (Sarantis et al., 1988).”

 

Page 293:  “In conclusion, antiprogestines such as mifepristone (RU 486) or ZK 98734 given in rats on days 1 and 2 post-coitum, can reduce the implantation rate to zero.”

 

Now you know what Planned Parenthood et al. have known for years.  Chemical contraceptives produce secondary contraceptive effects known as ABORTIONS!!!!!!  Now that you know the facts, you are no longer innocent but rather GUILTY OF MURDER if you continue your chemical birth control methods!!!!!!